Cirrhosis, Hyperammonemia and Hepatic Encephalopathy

By: Santiago GrisolÃa (Editor), Vicente Felipo (Editor)

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160 Pages

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25.4 x 17.78 x 1.12

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Details the brain damage that can result from high ammonia levels in the blood because of a damaged liver, and both traditional and proposed new methods for mitigating it, including nutritional approaches. The 12 papers are the lectures presented at a summer course in El Escorial, Spain, August 1992

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Non-Fiction Medicine Medicine in General Medical Equipment & Techniques Medical Laboratory Testing & Techniques Clinical & Internal Medicine Gastroenterology Hepatology Neurology & Clinical Neurophysiology Science Biology, Life Sciences Zoology & Animal Sciences

Botany & Plant Sciences

Control of Urea Synthesis and Ammonia Detoxification	p. 1
Brain Metabolism in Hepatic Encephalopathy and Hyperammonemia	p. 13
Ammonia Metabolism in Mammals: Interorgan Relationship	p. 21
Clinical Manifestations and Therapy of Hepatic Encephalopathy	p. 39
Nutritional Considerations in Patients with Hepatic Failure	p. 45
Do Benzodiazepine Ligands Contribute to Hepatic Encephalopathy?	p. 57
Effects of Hyperammonemia on Neuronal Function: NH[subscript 4][superscript +], IPSP and Cl[superscript -] Extrusion	p. 71
Activation of NMDA Receptor Mediates the Toxicity of Ammonia and the Effects of Ammonia on the Microtubule-Associated Protein MAP-2	p. 83
Modulation of the Exocytotic Release of Neurotransmitter Glutamate by Protein Kinase C	p. 95
Controls of Cerebral Protein Breakdown	p. 107
Two Different Families of NMDA Receptors in Mammalian Brain: Physiological Function and Role in Neuronal Development and Degeneration	p. 119
Glanglioside GMl and its Semisynthetic Lysogangliosides Reduce Glutamate Neurotoxicity by a Novel Mechanism	p. 129
Contributors	p. 143
Index	p. 145
Table of Contents provided by Blackwell. All Rights Reserved.

Cirrhosis, Hyperammonemia and Hepatic Encephalopathy

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Hardcover

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